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Intriguingly, the authors hypothesize progesterone amplification of this pathway through PGRMC1, noting E6s selective degradation of nuclear progesterone receptors (sparing membrane isoforms), which may sustain autophagy-mediated immune evasion in HPV oncogenesis [183, 184]

Factors which further affect premature aging include telomere deficiency, mitochondrial dysfunction, oxidative stress, DNA damage, and altered protein function