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When hearing loss occurs at a specific frequency, the brain may generate a sound to compensate for the missing signal

Clustering analysis confirmed the evident distinction between G93A-SOD1 and control epSPCs as shown in heatmaps of statistically significant metabolites between the two conditions

These findings contrast those of Dvl3 null mice, which displayed PCP defects in addition to loss of outer hair cells (Etheridge et al., Dvl3 null mice with one functional allele of Dvl2 exhibits more severe PCP defects than Dvl3 null animals alone, again suggesting Dvl members may complement each others

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