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Mechanistically, this redoxin directly reduces lipid hydroperoxides [180,181] , alleviates ferroptosis-induced mitophagy [182] , remodels membrane phospholipid composition [183] , or diminishes inflammatory pathways [173,176]

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Under the impact of risk factors such as elevated blood glucose, blood pressure, and cholesterol lipids, the vascular endothelium undergoes endothelial dysfunction, characterized by increased inflammation and oxidative stress, decreased NO biosynthesis, endothelial-mesenchymal transition, senescence, and even cell death

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