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Local overexpression of these genes in the mouse brain resulted in increased anxiety-like behaviour, while local inhibition of glyoxalase 1 expression by RNA interference decreased the anxiety-like behaviour

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53 This reveals a novel BCAA metabolism inhibition mode in the context of KRAS inhibition of BCAT2 ubiquitination degradation, explaining the preference for PDAC amino acid metabolism

In this context, the focus lies not on any direct peptide-to-muscle effect, but on how receptor-driven endocrine signaling remodels downstream muscle biology [6]