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Regulatory mechanisms of aging in aging-associated diseases The hallmarks of aging and aging-related diseases do not exist in isolation

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In the light of these findings, the functional plasticity of the intestinal epithelium and its regenerative response to injury are modulated or even controlled by mitochondrial metabolism, supporting the hypothesis that mitochondrial exhaustion contributes to the functional perturbation of the epithelium in IBD (referred to as metabolic injury)

In addition, the immune component of the barrier limits access of environmental antigens, while the biological component is maintained through colonization by commensal microbiota 70