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Moreover, decreasing the levels of intra-lysosomal cystine by cysteamine did not alter this mechanism and thus CTNS itself and not cystine is likely to be responsible for the activation of this pathway [38]

46 APAP can freely cross the selective maternal-fetal barrier of the placenta, after which it can then be metabolized by fetal hepatocytes, causing fetal hepatic necrosis if appropriate therapy with NAC is not administered in a timely manner

Nutritional metabolites are much cheaper than inhibitors, thus offering profit potential