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Research has demonstrated that mitophagy is activated, as evidenced by increased Parkin translocation to mitochondria, ubiquitination of mitochondrial proteins, sequestration of impaired mitochondria into autophagosomes, and degradation of mitochondrial proteins in primary mouse liver hepatocytes (Williams et al., 2015b

[DOI] [PMC free article] [PubMed] [Google Scholar] Liu Q., Zhao B

Molina-Vega M, Picn-Csar MJ, Gutirrez-Repiso C, Fernndez-Valero A, Lima-Rubio F, Gonzlez-Romero S, et al

In late-stage CKD, ferroptosis and necroptosis form a positive feedback loop: ferroptosis-derived DAMPs (e.g., HMGB1) activate RIPK3MLKL signaling, while necroptosis releases labile iron, further fueling ferroptosis