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Within this framework, iron accumulation may signal heightened susceptibility to ferroptosis, reflecting defective iron mobilisation and organellar crosstalk rather than simply a reservoir of pro-oxidant potential

Vitamin C is essential for collagen cross-linking

Chronic inflammation may interact with skeletal muscle iron overload, aggravating muscle atrophy

Through systematic identification of pivotal hub genes (NQO1, AURKA, and CDKN2A), this study has not only elucidated the synergistic mechanisms underlying the ferroptosis-cuproptosis regulatory network in HNSCC progression, but also characterized the clinically resistant Epi_2 molecular subtype with distinct therapeutic vulnerabilities