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The expression levels of (h)GSH biosynthesis-related genes, defense response genes ( PR1 and PR10 ), and that of the nodule senescence marker gene Cyp15a were not significantly changed in roots with nodules of GSH-treated wild-type plants (Figure 10)

Although GSH efflux may be a mechanism to circumvent the normally protective role of GSH, it appears essential for apoptosis to occur in many cell types [4, 36]

Tobias P Dick 2 Marcus Gutscher 2 , Anne-Laure Pauleau 2 , Laurent Marty 3 , Thorsten Brach 3 , Guido H Wabnitz 1 , Yvonne Samstag 1 , Andreas J Meyer 3 , Tobias P Dick 2 Article | DOI: 10.1038/nmeth.1212 Affiliations: Institute of Immunology, University of Heidelberg, Heidelberg, Germany Redox Regulation Research Group, German Cancer Research Center (DKFZ/A160), Heidelberg, Germany Heidelberg Institute of Plant Sciences, University of Heidelberg, Heidelberg, Germany Less Citations: 522 Access enabled via: An Institution Abstract Dynamic analysis of redox-based processes in living cells is now restricted by the lack of appropriate redox biosensors