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This suggests that Grx1 may regulate TDP-43 protein stability through mechanistically distinct pathways, potentially involving redox-dependent posttranslational modifications such as S-glutathionylation that directly influence TDP-43 protein stability

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Iron accumulation and lipid peroxidation may be considered as intermediate events but they are not the final executors of ferroptosis

Various in vitro and in vivo studies have confirmed the antidiabetic, hypolipidemic, hypotensive, neuroprotective, and cardioprotective properties