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Activation of nuclear factor-B (NF-B) induces transcription of target genes that primarily encode survival proteins (e.g., cellular FLICE-like inhibitory protein and inhibitor of apoptosis proteins) via formation of the complex I signalosome after binding to TNF receptor-1

Quercetin plus cisplatin worked synergistically to suppress the growth of cultured hepatocellular carcinoma cells (Zhao 2014), and quercetin enhanced the ability of 5-FU to inhibit cell growth and stimulate apoptosis in esophageal cancer cells (Chuang-Xin 2012)

The anti-inflammatory pathways are primarily mediated by the farnesoid X receptor (FXR) and the Takeda G protein-coupled receptor 5 (TGR5) 147

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