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Significant progress has been made in constructing GSH-responsive drug delivery systems (DDSs) capable of site-specific therapeutic release within the tumor microenvironment [15]

The sensitive peptide-bound sp 2 carbonyl group in the transition state is converted into a sp 3 secondary alcohol within the gem-diol intermediate, ultimately leading to peptide bond loss during this transitional phase (Fig

Downstream of mTORC2 AKT, a key substrate of mTORC2, regulates glucose metabolism by inhibiting glycogen synthase kinase 3 (GSK3) and the Forkhead Box O (FOXO) family of transcription factors

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