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Complement complexes can be discovered in atherosclerotic lesions, and ECs exposed to complement are activated and improves the production of IL-6, IL-8 and MCP-1, thereby triggering the adhesion of monocytes to ECs 89

Large-scale randomized controlled trials are urgently needed to establish safe lipid thresholds and optimize therapeutic strategies

It was found that a 23-fold -synuclein overexpression in hippocampal and ventral midbrain neurons impeded synaptic vesicle release [116] while -synuclein knock-out mice displayed stronger release of DA upon stimulus [117]