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During differentiation, PKC localizes in the nucleus and blocks Hmga1 gene expression to promote myoblast formation [162]

Forty-four percent of participants in the study experienced full remission of all BMS symptoms after treatment (x)

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Transcriptomics analyses of unc1 mutants revealed changes in the expression of genes involved in fat metabolism, suggesting that fat metabolism changes, controlled by neurohormonal signalling, contribute to protein homeostasis