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Fluoxetine-induced androgen deficiency may lead to the increased expression of ubiquitin carboxyl-terminal hydrolase L1 (UCHL1), which activates the pro-apoptotic factor p53 through deubiquitination and triggers germ cell apoptosis

On the one hand, it appears to induce oxidative damage and cell death, and high levels of 3-HK are associated with several psychiatric disorders

10.1385/0-89603-268-X:9 38 LeeB

Bypass signaling activation Cellular plasticity, governed by balanced regulatory networks involving feedback loops, isoform switching and pathway crosstalk, enables DTPs to evade targeted therapies through compensatory signaling