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reported that matrix detachment of lung cancer cells induces a decrease in glucose uptake, activates LKB1 and AMPK, inhibits Ac-CoA carboxylases 1 and 2 which in turn, decreases NADPH consumption in fatty acid synthesis (FAS) while increasing NADPH generation through FAO fueling [118]

PERK is required at the ER-mitochondrial contact sites to convey apoptosis after ROS-based ER stress