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these include: Underlying bacterial infection or toxin-related rash (eg, staphylococcal scalded skin syndrome and toxic shock syndrome) Viral exanthems, including measles, rubella, human immunodeficiency virus (HIV), roseola due to human herpes-virus 6, Epstein-Barr virus and many others Irritant contact dermatitis Folliculitis Sweat-induced miliaria or transient acantholytic dermatosis (Grover disease) Erythema multiforme due to non-drug causes Idiopathic generalised pustular psoriasis (AGEP may mimic this) Non-drug-induced cutaneous lupus erythematosus Autoimmune blistering diseases Other causes of erythroderma (eg, eczema, psoriasis, and lymphoma)

.Gmez-SerranoM.MarinaroF.LpezE.PulidoM.PreuerC.et al (2021)

After the model was expanded to include GS-H 2 Q adduct formation as a function of its reduction potential, it was then combined with the Reed glutathione model 21 in order to simulate how the quinone redox-metabolism of doxorubicin influenced the glutathione metabolism

Reasons include: reliance on animal studies anecdotal reports limited human data commercial marketing Scientific conclusions should rely primarily on peer-reviewed evidence