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Discussion Here, we present the first evidence of the physiological role of endogenous hepsin in redox homeostasis and its potential protective effect early after APAP exposure, which is crucial for mitigating liver injury

doi: 10.3233/rnn-201131 117

Therefore, putative mechanisms include lingering damage from direct viral infection of cardiomyocytes and endothelial cells and viral persistence in the CV system (70, 71)