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Keap1 forms part of an E3-ligase complex and promotes continuous proteasome-dependent degradation of Nrf2, thereby suppressing its activity (53, 54)

Hepatic fat accumulation results from an imbalance between lipid acquisition and lipid disposal, which are regulated through four major pathways: uptake of circulating lipids, de novo lipogenesis (DNL), fatty acid oxidation (FAO), and export of lipids in very low-density lipoproteins (VLDL) (Fig

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