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In addition, these reactive metabolites can bind to proteins, lipids, or nucleic acids and inhibit the enzymes in the mitochondrial ETC, resulting in oxidative stress, mitochondrial dysfunction, and hepatotoxicity [310, 311]

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This may have important clinical considerations given that FHd-RCC is managed as a non-clear cell RCC under the European Society for Molecular Oncology (ESMO) guidelines, which recommend first-line antiangiogenic therapy 4

The recent studies in melanoma and colon cancer murine syngeneic tumor models demonstrated that tumor cells and immune cell populations such as antigen-presenting conventional dendritic cells (cDC) responsible for the priming of the CD8+ cytotoxic T cells and CD4+ T cells are competing for Gln uptake