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GSH plays an essential role in cell signaling and defense by functioning as an enzyme cofactor, modulating protein function via glutathionylation, and detoxifying reactive metabolites and xenobiotics (18,19)

Similar to the adult rat TBI model study by Shuhmann et al., we observed significant decrease in concentrations of several neurometabolites in the ipsilateral hippocampus at 24 h post TBI

In estrogen-deficient states, increased oxidative lipid remodelling coincides with impaired antioxidant defenses, driving chronic inflammation and atherogenic lipid profiles

CBD acts as an agonist of the receptors/channels TRPA1, TRPV1, TRPV2, TRPV3, PPAR, 5-HT1A, and A1A, and as an antagonist of the receptors GPR55, GPR18, and 5HT3A (see Table 2 for references)