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It was reported that edaravone inhibited ferroptosis by scavenging chain-initiating water-soluble radicals and chain-carrying lipid peroxyl radicals due to its amphiphilicity, in ischemic stroke and amyotrophic lateral sclerosis ( N -acetylcysteine reduced neuronal ferroptosis and improved behavior following intracerebral hemorrhage in mice through neutralizing nuclear ALOX5-derived toxic lipid species which is involved in the cellular lipid synthesis process and is needed for ferroptosis ( The relationship between ferroptosis and other forms of cell death is unknown