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Since semaglutide further slows gastric emptying as part of its mechanism of action, patients with pre-existing gastroparesis may experience worsening of symptoms including nausea, vomiting, bloating, and abdominal discomfort

1,48 According to WJWEs efficacy in mitigating DHT-induced follicular miniaturization and oxidative stress in our AGA model, we proposed that it may also benefit to FAGA patients by targeting these pathways

This regulated necrotic cell death is activated in response to Fas/FasL signaling, CD59 ligation combined with pore formation, mediated by ROS, ceramide, and advanced glycation end products (AGEs) [28]

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