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Mechanistically, Kla modification orchestrates TAM-dependent tumor progression through tissue-specific pathways: in gastric cancer, H3K18la induces sustained VCAM1 expression to activate AKT/mTOR/CXCL1 signaling and facilitate M2 macrophage recruitment (72)

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The results demonstrated that CAR-T, which has enhanced mitochondrial OXPHOS that utilizes fatty acids and reduced glycolytic activity, demonstrated more robust tumor elimination [115]