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Maturation of nave T cells depends on autophagy to reduce mitochondrial and ER contents through mitophagy and reticulophagy, respectively (143, 145, 146)

Therefore, in this study, we tested the hypothesis that administration of DHEA may alleviate APAP-induced hepatic damage by attenuating oxidative stress, inflammation, and apoptosis through stimulating SIRT1

10.1038/s41584-023-00947-z 125 PradeepC

With mutagenesis studies, we propose an acyl-modification driven ghrelin-binding model, in which the binding of octanoyl group in cavity II orients the N-terminus of ghrelin to cavity I, and leads to the initiation of signal transduction