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However, futile quinone redox cycling can lead to a cascade of ROS formation and as such, the link between toxicity and quinone redox cycling is widely acknowledged 5

On the other hand, macrophages, overexpressing integrin 4, can interact with overexpressed vascular cell adhesion molecule-1 (VCAM-1) on lung cancer cells and be efficiently recruited to the tumor region by the chemokine C-C motif ligand 2 (CCL2), making macrophage membrane-based nanovesicles excellent carriers for enhanced delivery to lung tumors [17,18,19,20]

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