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These lesions were observed in the cerebrum, cerebellum, and meninges indicating the induction caused diffuse brain injury (Figures 1416)

Chen D, Zhang T, Lee TH (2020a) Cellular mechanisms of melatonin: insight from neurodegenerative diseases

Am J Ind Med, 14: 25-36

In addition, tumor necrosis factor (TNF) binding to TNFRSF1B activates TRAF2/MAPK signaling, enhances TRPV1 and SCN10A expression, and thereby facilitates NPP development in sequence (134)