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In SOD1 G93A mice, a ketogenic diet was shown to protect against the loss of motor neurons in the spinal cord and to delay the onset of behavioral symptoms (Zhao et al., 2006)

Mice expressing adipocyte NDI1 displayed similar weight gain, activity, caloric intake, energy expenditure, fat pad weights and body composition during HFD feeding compared with control mice (Fig

It was observed that pro-inflammatory cytokines enhance CHI3L1 expression in adipocytes, suggesting a strong association between CHI3L1 and obesity, as well as obesity-induced pro-inflammatory responses ( The observation of a bioactive lipid, sphingosine-1-phosphate (S1P), demonstrated potent regulation of the expression of several genes involved in the control of cell proliferation and airway remodeling ( HBEGF, TGFB3, TXNIP, PLAUR, SERPINE1, RGS4 ) ( The association between genetic predisposition and obesity-related airway remodeling intricately involves the role of adipokines

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