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193 In addition, Fusobacterium nucleatum can activate the RIPK1-mediated apoptosis pathway in UC, resulting in the destruction of tight junctions between cells, which in turn promotes the destruction of the intestinal barrier, inhibits the renewal of intestinal epithelial cells, and accelerates the progression of UC

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ii) pathologies that increase oxidative stress such as diabetes or other metabolic diseases (dyslipidemia, in case of group I and III), atherosclerosis (diagnosed), hypertension, autoimmune diseases, allergies, asthma, hematopoietic dysfunction, infectious diseases, oncological pathology, ulcer gastro-duodenal, acute pancreatitis and Parkinson's disease

Kumar A, Dubey AK, Kumar V et al (2020a) Over-expression of chickpea glutaredoxin (CaGrx) provides tolerance to heavy metals by reducing metal accumulation and improved physiological and antioxidant defence system