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Representative systems include Fe-HMME@DHA@MPN

(222) observed that a lack of GPx4 can promote the production of TGF-1, thereby exacerbating fibrosis, while upregulation of GPx4 can reverse this change

Koren) 59 (Motherisk, 2000)

Indeed, several studies have gathered evidence of the intricate interactions between skin-resident microbes and host cells in shaping a disease-promoting environment [19, 20]