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Furthermore, Doxorubicin and Cyclophosphamide reduced synaptic plasticity via hindering expression of brain-derived neurotrophic factor, phosphorylated cyclase response element binding protein, synaptophysin, and postsynaptic density protein 95 whereas protein expression of such synaptic plasticity biomarkers was enhanced by L-carnitine treatment

As caffeine mimics the effect of dopamine on striatopallidal neurons, it causes a progressive sensitisation of cannabinoid CB1 receptors, which control GABAergic inhibitory postsynaptic currents (IPSCs) [115]