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Li G, Ko CN, Li D et al (2021) A small molecule HIF-1 stabilizer that accelerates diabetic wound healing

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R HDAC inhibition, NF-kB, and the epigenetic basis of anti-inflammatory signaling: When inflamed colonocytes receive excess butyrate that cannot be fully oxidized (due to mitochondrial dysfunction in the inflammatory state), unmetabolized butyrate accumulates and inhibits class I and class II HDACs