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Abstract Studies in the last two decades have firmly established that the gaseous free radical nitric oxide (NO) is an intracellular and intercellular mediator of signal transduction pathways controlling plant growth and development, as well as plant responses to biotic and abiotic stresses

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This indicates that, despite the high correlation of promoter methylation with ACB expression and drug sensitivity, epigenetic silencing is not a primary cause for the lack of ACB induction by NRF2 and the inability of SCLC to adapt to drug-induced ROS stress

Furthermore, insulin excess amplifies hypothalamic gonadotropin-releasing hormone (GnRH) pulsatility, elevating LH and further driving ovarian androgen production