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Abstract Interfering with interactions of vascular endothelial growth factors (VEGFs) with their receptors (VEGFRs) effectively inhibits angiogenesis and tumor growth

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5,8,9,10,11,12,13,14,15,16,17,18,20 The widespread clinical use of potent second-generation ARPIs has further expanded AR -/lo foci, which now comprise up to 2030% of metastatic CRPC cases