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Anti-amyloid antibodies (lecanemab, donanemab) are monoclonal antibodies that bind and clear amyloid-beta, the protein that aggregates into plaques in Alzheimers disease, and they are the clearest disease-modifying drugs to reach the clinic

These results suggest that reduced homocysteine causes protein thiol homocysteinylation that may induce smooth muscle cell proliferation by binding to a redox receptor and that may exacerbate atherosclerotic lesion formation by inducing collagen expression (Tyagi, 1998)

is a clinical research investigator Bristol Myers Squibb and Takeda

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